BYU ScholarsArchive

Title

The Role of a Mutated Receptor, CCRL2 and its Correlation with a Risk Factor for Alzheimer’s Disease

Authors

Colby J. Hendrix, Brigham Young University - ProvoFollow
Christopher J. Haynie, Brigham Young University - Provo
Hunter G. Lindsay, Brigham Young University - Provo
Josue D. Gonzalez, Brigham Young University - Provo
Scott Weber, Brigham Young University - ProvoFollow

Files

Keywords

Alzheimer's Disease; Neuroinflammation; Chemokines

Abstract

Alzheimer’s Disease ( is the most common cause of dementia, which is the 5 th leading cause of death While the exact cause of AD is unknown, many suggest that inflammation could be a culprit 1 Inflammation is driven by our immune system A key component of inflammation is chemokine signaling These signaling molecules help guide immune cells to areas of infection and damage Cells use chemokine receptors to interact with secreted chemokines (Fig 1 Left) Some chemokine receptors are identified as atypical chemokine receptors ( because they have different functions than conventional chemokine receptors in the immune system (Fig 1 One of these ACKRs, CCRL 2 is the focus of our work

BYU ScholarsArchive Citation

Hendrix, Colby J.; Haynie, Christopher J.; Lindsay, Hunter G.; Gonzalez, Josue D.; and Weber, Scott, "The Role of a Mutated Receptor, CCRL2 and its Correlation with a Risk Factor for Alzheimer’s Disease" (2023). Library/Life Sciences Undergraduate Poster Competition 2023. 3.
https://scholarsarchive.byu.edu/library_studentposters_2023/3

Document Type

Poster

Publication Date

2023-03-01

Language

English

College

Life Sciences

Department

Microbiology and Molecular Biology

University Standing at Time of Publication

Junior

Copyright Use Information

https://lib.byu.edu/about/copyright/