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Journal of Undergraduate Research

Keywords

selenium, NF-B, prostate cancer cells, cancer, Nuclear-Factor-kappa

College

Life Sciences

Department

Nutrition, Dietetics, and Food Science

Abstract

The American Cancer Society estimates that prostate cancer will account for 30% of new cancers in men, totaling over 189,000 new cases, with 30,200 deaths in 2002 (1). Because of its high prevalence and long latency time prostate cancer is an ideal candidate for chemoprevention (2). Recent research suggests an inverse relationship between high dietary selenium (Se) intake or status and risk for prostate cancer. However, the mechanisms for Se’s protective effects against prostate cancer have not yet been determined. This project focused sharply on a single mechanism by which Se may alter gene expression and affect risk for prostate cancer. Demonstration of this mechanism could suggest other interventions, which may be effective in reducing prostate cancer risk by targeting the same processes.

Included in

Nutrition Commons

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