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Journal of Undergraduate Research

Keywords

GABA(A) receptors, drug dependence, dopamine-dependent pathways, alcohol addiction

College

Family, Home, and Social Sciences

Department

Psychology

Abstract

The prevailing model for addiction proposes that neurotransmission of dopamine (DA) in the brain reward system is dysregulated, with enhanced levels during acute drug exposure and reduced levels during drug dependence and withdrawal. One emerging view is that during drug addiction, GABA(A) receptors switch the effect of GABA neurons on DA neurons from inhibitory to excitatory. This has been demonstrated for opiate addiction (Vargas-Perez et al., 2009). Our lab has previously demonstrated that GABA neurons become tolerant to acute ethanol and hyperexcitable during chronic ethanol (Gallegos et al., 1999), which may explain why dopamine neurotransmission is reduced during withdrawal. The aim of the present study was to evaluate the effects of GABA(A) receptor agonists on the firing rate and GABA synaptic transmission to other GABA neurons associated with alcohol dependence.

Included in

Psychology Commons

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