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Keywords

Anxiety, Subclinical, Treatment, Neurobiology

Abstract

Generalized Anxiety Disorder (GAD) is one of the leading mental illnesses in the United States today (Alvarez et al., 2012). However, a large number of individuals have their lives disrupted by the symptoms of anxiety, but their symptoms are not severe enough to be diagnosed with GAD. These individuals, much like individuals with GAD, have high trait anxiety, differential brain structure and function, and hypervigilant performance monitoring. Further understanding the neural correlates related to subclinical generalized anxiety disorder and how the neural mechanisms involved relate to daily functioning is of utmost importance. Since there are individuals suffering from subclinical anxiety symptoms on a day to day basis, it may be beneficial to have treatment options that are open to all in order to alleviate the disruption that anxiety can cause in daily functioning.

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